Atypical Diabetes: Pathophysiology, Clinical Presentations, and Treatment Options by Boris Draznin
- Atypical Diabetes: Pathophysiology, Clinical Presentations, and Treatment Options
- Boris Draznin
- Page: 560
- Format: pdf, ePub, mobi, fb2
- ISBN: 9781580406666
- Publisher: American Diabetes Association
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Atypical Diabetes: Pathophysiology, Clinical Presentations, and Treatment Options by Boris Draznin Although the vast majority of diabetes cases (type 1 and type 2) are polygenic diseases, research, funded by the American Diabetes Association, has found that some forms of diabetes, distinct from type 1 or type 2 diabetes, are caused by single gene mutations. It has been estimated that these monogenic forms of diabetes account for up to five percent of all diabetes cases. In addition to monogenic diabetes, there are other atypical causes of diabetes, including: genetic defects in insulin action; diseases of the exocrine pancreas; and endocrinopathies. Given recent advances in our understanding of sub-types of diabetes, the time has come for a book on how to correctly diagnose and treat patients with atypical forms of this disorder. The book will be divided into three parts, each exploring distinct categories of atypical diabetes. Part I: Monogenic Diabetes: Genetic Defects of β-cell development and function; Part II: Insulin resistance, genetic defects in insulin action, and diseases of exocrine pancreas; Part III: Endocrinopathies, Immune-mediated diabetes, Diabetes of Unknown cause, and Other genetic syndromes sometimes associated with diabetes.
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Diabetes diagnosis, pathophysiology, and treatments in SMi Source. Update on diagnosis, pathogenesis and - Semantic Scholar
clinical presentation has been reported primarily in African–Americans (AA) and Latinos [6,7,9,15], but also in other minority ethnic groups [13,16–18]. This variant of T2DM has been referred to in the literature as idiopathic T1DM, atypical dia‑ betes, Flatbush diabetes, diabetes Type 1½ and more recently as ketosis‑prone Adult-Onset Atypical (Type 1) Diabetes - Semantic Scholar
autoantibodies and fluctuating insulinopenia). RESEARCH DESIGN ANDMETHODS — Eight patients with atypical type 1 diabetes ments and ketosis- prone episodes, as well as clinical features of type 2 diabetes. Only one subgroup could be strictly Diabetes Care 27:1108 –1114, 2004. In 1997, the American Diabetes Atypical Diabetes : Pathophysiology, Clinical Presentations, and
Find product information, ratings and reviews for Atypical Diabetes :Pathophysiology, Clinical Presentations, and Treatment Options (Paperback) ( Boris online on Target.com. Improved Glycemic Control in Subjects with Atypical Diabetes
with diabetic ketoacidosis (DKA) as the initial manifestation treatment. We hypothesized that recovery from atypical di- abetes resulted from a restoration of Я-cell function and improved insulin sensitivity. Subjects and Methods. Experimental subjects. The study was performed in General Clinical ResearchClinic. (GCRC) PRIMARY CARE OBJECTIVES
diagnostic entities outlined below in terms of clinical presentation, diagnostic work-‐ up and management. Diagnose presentations of ischemic heart disease that are classic and atypical. (e.g, women, diabetes, young Acneiform Conditions. 1. Describe the different stages of acne vulgaris and discusstreatment options. American Diabetes Association Books | List of books by author
See all books authored by American Diabetes Association, including American Diabetes Association Complete Guide to Diabetes, and Diabetes A to Z : What You Need to Know About Diabetes--Simply Put, and more on ThriftBooks.com. Atypical Diabetes: Pathophysiology, Clinical Presentations, and TreatmentOptions. Ketosis-Prone Type 2 Diabetes in Patients of Sub-Saharan African
Detailed assessment of medical history, symptoms of hyperglycemia/insulin deficiency (thirst, polyuria, polydipsia, and weight loss), and precipitating cause of at the outpatient diabetes clinic of the previously cited hospitals, where blood glucose, HbA1c, lipids, and anthropometric parameters were determined every 3 –6